Se você está começando agora a trabalhar pela internet, provavelmente está pesquisando sobre quais são os melhores produtos para vender como afiliado, por isso hoje eu quero te dar algumas dicas certeiras que me ajudaram a se destacar da maioria.

A ordem que estou mostrando abaixo não quer dizer necessariamente que precisa ser exatamente nesta ordem, você pode escolher por qual deles começar.

1- Produtos recorrentes

Escolha pelo menos um produto com comissões recorrentes para você trabalhar, um produto com comissões recorrentes te paga pela venda e também na recorrência, seja mensal, semestral ou anual.

Se você escolher um produto recorrente nas plataformas de afiliados já vem informando no próprio produto quanto você ganha na venda e quanto você ganha na recorrência, ou seja, você vende apenas uma vez e pode ganhar por anos.

A grande maioria destes produtos tem comissões mais baixas, então não seja imediatista, pense a longo prazo, em construir uma base de leads recorrentes trabalhando como afiliado.

Ao pesquisar quais os melhores produtos para vender como afiliado você pode no primeiro momento desprezar estes produtos recorrentes (assinaturas) mas se você pensar com calma vai perceber que você pode ganhar muito mais dinheiro como afiliado com muito menos esforço.

Veja um exemplo de apenas um dos produtos que promovo como AFILIADO da Hotmart.

 

Eu vendi como AFILIADO, ou seja, um produto de terceiros que nem conheço pessoalmente, a venda foi feita a 35 meses atrás e estou ganhando até hoje SEM FAZER NADA!

Claro que não cabe tudo na tela, tem várias outras vendas deste produto e de outros.

Talvez você ache que isso é uma mixaria, e de fato é! Mas multiplique isso por 100, 300, 1000 vendas recorrentes todos os meses, sabe aqueles dias de vagas magras? Ou aquele período de final de ano que você nem está se dedicando ao seu negócio?

Está em viagem de férias com sua família e quando pensa que não, o aplicativo da plataforma (Hotmart, Eduzz, Monetizze) toca te avisando que você vendeu!

Isso é recorrência meu amigo! É para quem pensa grande a longo prazo, imagine você sendo afiliado de produtos assim? Divulgando da forma correta, trabalhando pela internet de forma honesta e séria.

Você acorda pela manhã tendo a certeza absoluta que vai ganhar dinheiro pela internet mesmo se não trabalhar, se você falar isso para uma pessoa leiga ela vai achar que você está mentindo, pois não entende de onde vem o dinheiro.




Existem muitos produtos recorrentes para você vender como afiliado, basta você acessar as plataformas de afiliados e fazer uma pesquisa minuciosa, você vai ver que tem produtos assim em vários nichos como:

  • Teologia
  • Música
  • Marketing
  • Saúde
  • Arte
  • Gastronomia

Entre outros…

2- Produtos Físicos

É comum quando um afiliado está começando a trabalhar pela internet já querer começar se afiliando a algum curso de marketing digital, muitos têm a ilusão que só é possível ganhar dinheiro como afiliado vendendo cursos que ensinam a vender como afiliado.

Mas isso é um grande erro, o mercado de afiliados é muito maior do que formulas e formulas de ganhar dinheiro pela internet, é preciso aprender como vender, depois que você aprende como vender de fato você consegue vender quase tudo pela internet.

Vender produtos físicos como afiliado é um mercado pouco explorado pela maioria dos afiliados, existem muitos produtos bons nas plataformas de afiliados como a Eduzz, Monetizze, Braip, Awin, Lomadee, Aliexpress entre muitas outras.

Mesmo você sendo pessoa física é possível começar a vender produtos físicos como afiliado, inclusive tem aulas sobre isso dentro do meu portal de membros CLUBE SUA META.

Vender produtos físicos como afiliado te permite em muitos casos nadar em um oceano muito mais azul, como por exemplo através de parcerias com influenciadores digitais é possível conseguir um tráfego grande em minutos e muitas vendas.

Escolha produtos físicos com menor concorrência, se você está começando agora e não vai investir em anúncios pagos fuja dos produtos de sexualidade da Monetizze por exemplo, estes produtos tem uma concorrência bem grande e é mais indicado para quem não tem vergonha de fazer vídeos por exemplo, ou para quem tem dinheiro para arriscar no tráfego pago.

3- Produtos verdadeiros

Pode parecer um pouco obvio, mas muitos produtos não são verdadeiros, existem produtos na Monetizze por exemplo que promete um aumento peniano em alguns dias apenas um gel “milagroso” se você fizer uma busca rápida na internet vai ver um monte de reclamação de homens que usaram e não tiveram nenhum resultado.

Assim como em outros nichos, existem cursos que te prometem ganhar dinheiro pela internet em poucos dias, fazer cem mil em sete dias, mas na verdade isso acaba não acontecendo.

Então ao escolher um produto para divulgar, pesquise sobre o autor, sobre o produto, depoimentos reais, veja se de fato tem lógica o que está sendo proposto ou é apenas um copy forte para vender mais.




Antes de você definir o produto pense primeiro como você irá promover aquele produto, ou seja, se você escolher um produto de ganhar dinheiro pela internet você irá concorrer com pessoas que fazem vídeos no Youtube todos os dias, você está disposto a isso?

Ou você é do tipo tímido que não quer se expor?

Se você se encaixa neste segundo perfil nem vai para um nicho assim, escolha um nicho que você possa vender sem se expor, onde não tenha tantas “estrelas” experientes divulgando aquele produto ou nicho.

Veja o exemplo do meu aluno Fabiano por exemplo, ele vende todos os dias pela internet, já é meu aluno a anos, mas não fica focado neste nicho de marketing digital, ele vende desde cursos de teologia, enfermagem, brigadeiro gourmet entre outros.

 




É importante você entender que trabalhar como afiliado é algo sério, apesar de muita gente sem noção passar uma imagem de dinheiro rápido e fácil.

Quando alguém me pergunta quais os melhores produtos para vender como afiliado eu sempre falo que depende, depende de como ela irá divulgar, quais os objetivos da pessoa se é renda extra ou quer de fato mudar de vida e ganhar muito dinheiro.

Levar este negócio de forma profissional, estudar, aplicar e persistir, este é o “segredo” para vender bem todos os dias, até mesmo quando não estiver trabalhando.

Quer aprender desde o básico? Conheça o fantástico CLUBE SUA META e tenha acesso a todos os meus treinamentos em vídeo aulas passo a passo com 50% de desconto.

 

 

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  3. Prior to sacrifice, mice had been deeply anesthetized with sodium pentobarbital (90 mg/kg) and chlorohydrate (120 mg/kg).

    The soleus was surgically eliminated and placed in a modified Kreb’s
    resolution with pH 7.four, at 37 °C, gassed with 95% O2/5% CO2,
    and circulated at 10 mL/min. Muscle Tissue, spanned
    by parallel platinum plate electrodes, have been mounted horizontally utilizing #5–0 silk sutures with one hook hooked up to an isometric Harvard-Hugo Sachs F30 drive transducer (Harvard Equipment,
    Holliston, MA). Muscular Tissues were adjusted to optimum
    resting length (Lo) to acquire maximum twitch pressure.
    Stimulation amplitude, at the supramaximal threshold stage of 80 mA, consisted of either a single pulse (twitch
    response, zero.2 ms) or defined pulse trains (tetanus response, Po;
    0.2 ms pulse width, one hundred forty Hz, 500 ms).

    The indications being targeted embrace varied muscular diseases and disorders, corresponding to Spinal Muscular Atrophy, Muscular Dystrophy, Muscular
    Atrophy, and Neuromuscular Illnesses. The most rapidly progressing drug types are monoclonal antibodies and fusion proteins, indicating intense competition within the subject.
    The growth of medication underneath the goal myostatin is spread
    across multiple countries, with the United States leading by way of the number of
    drugs in numerous stages of improvement. General, the lengthy run development
    of goal myostatin exhibits promise in addressing unmet medical wants within the field of muscular diseases and disorders.
    The mechanism of motion of MSTN relies on its interaction with
    different proteins within the pathway leading to the
    transformation of latent MSTN to its mature form and further activation of the
    Smad pathway, which finally ends up in the inhibition of myogenesis.
    Studies have proven PPIs are key mediators of assorted signaling and regulatory
    networks (Villoutreix et al., 2008).
    In addition, the mRNA and protein of FMOD, MSTN, and
    adipogenic markers (CD36, PPARγ, and CD163) were significantly decreased
    in Ac-MIF1-treated cells, and FMOD and PPARγ mRNA and protein expression had been decreased in Ac-MIF2-NH2 (Figure 7B).
    Furthermore, Smad2 and Smad3 mRNA expression was decreased in Ac-MIF-treated cells (Supplementary Determine
    S12). MSTN protein effects have been observed in C2C12 cells after 2 days of remedy in proliferation or myogenic differentiation media.
    Cell proliferation and fusion indices have been reduced by MSTN protein treatment
    (Supplementary Figure S1).
    The different drawback was that Welle believed adults would wish to
    proceed taking such dietary supplements in the occasion that they needed to maintain their
    positive aspects. More brittle tendons increase the probabilities of tissues snapping/rupturing, which,
    as you’ll have the ability to imagine, can be excruciatingly painful.
    He concluded that something which decreases myostatin by 60% or less is just about ineffective (if your goal is to build muscle).

    After all, without BALCO’s examine, Flex and the opposite bodybuilders taking
    part wouldn’t have known any completely different. Not all Mr Olympia bodybuilders have been tested for such mutations,
    however a few of them may actually have comparable genetics without even understanding it.

    Resistance training and common moderate-intensity cardio train are confirmed efficient natural methods for potentially reducing myostatin ranges and
    supporting muscle growth. While some medication and supplements declare to have an result on myostatin ranges,
    their actual impact alone on muscle development ought to be seen with a healthy dose of skepticism.

    If you’re dedicated to constructing lean muscle mass, you will comprehend it takes much effort—hours at the health club and a protein-rich
    diet are normally the recipe. But for many who have already dialed in their workout routines and
    nutrition and are looking for an additional edge, understanding how myostatin inhibitors
    work would possibly help you reach the following step in achieving
    these health targets. Epi 2.0 by Vital Alchemy offers a targeted strategy to enhancing muscle progress by inhibiting myostatin, facilitating improved muscle dimension and power.
    Quercetin is a flavonoid that acts as a robust antioxidant and also has potential myostatin inhibiting properties.
    It helps endurance and performance by improving blood move and reducing irritation.
    The failure of ACE-083 to satisfy scientific endpoints ultimately led Acceleron to discontinue its improvement and shift its focus of TGF-β concentrating on therapeutics away from muscle-dystrophic illnesses.
    Myostatin; also called development differentiation issue
    eight (GDF8) has been nicely reported to negatively regulate muscle development and measurement (Carnac et
    al., 2007; Chen et al., 2021). The putative involvement of MSTN in muscle atrophy has been documented in a
    quantity of research, prompting curiosity in MSTN as a therapeutic target to
    counteract muscle loss in patients with a variety of muscle-wasting situations (Baczek et al., 2020; Sartori et al., 2021).

    Similar studies measuring cardio and anaerobic capability in humans whereas supplementing (–)-epicatechin are limited.
    The outcomes of this study point out no benefit of (–)-epicatechin supplementation at 200
    mg per day at the facet of cycling. Moreover, (–)-epicatechin appeared to
    inhibit adaptations in relative peak cardio power and skeletal muscle SDH protein content,
    in contrast with the placebo. In conclusion, continual (–)-epicatechin supplementation mixed with
    train training in wholesome adults could additionally be
    disadvantageous. Extra analysis is needed to determine whether or not
    (–)-epicatechin supplementation would elicit a benefit with
    completely different dosing strategies or different
    train modalities. The failure of MSTN inhibitors to effectively treat muscle wasting
    illnesses in humans despite promising ends in preclinical studies could stem from species-specific variations in serum MSTN ranges.

    On common, human serum MSTN ranges are round 5–10 ng/ml, whereas mice
    exhibit concentrations exceeding 100 ng/ml, as much as a 20-fold difference [105, 106].

    Sarcoplasmic hypertrophy is when glycogen storage will increase the dimensions
    of the muscle cell, making it primarily temporary progress, that means if the myostatin level have been to return to normal, the glycogen within the
    muscle would decrease and mass could be misplaced.

    Venous blood from the antecubital vein was collected right into
    a serum separator tube utilizing a Vacutainer apparatus and needle
    (Becton, Dickinson and Firm, Franklin lakes, NJ).
    Immediately after blood draw, blood samples remained at room
    temperature for 20 min to clot, and then have
    been centrifuged (1,000 g) for 20 min to separate serum.
    Serum samples had been aliquoted into 1.5 mL tubes and instantly frozen at −80°C for the
    later evaluation. Percutaneous muscle biopsies (20–25 mg) had been obtained from the center portion of the vastus lateralis muscle on the midpoint between the patella and
    the higher trochanter of the femur at a depth between 1 and
    a pair of cm primarily based on previously-used procedures (20).
    The similar leg and common location (determined by pre-biopsy markings)
    was biopsied at each testing session.
    Myostatin (MSTN), which is referred to as a myokine, belongs to the
    remodeling growth factor β (TGF-β) superfamily and,
    as talked about above, acts as a unfavorable regulator of muscle progress.
    Serum MSTN has been reported to be a significant danger factor
    of pre-sarcopenia and sarcopenia [14,15], whereas MSTN
    knockout (MSTN−/−) mice exhibited larger myofiber dimension, muscle weight, and grip energy than wild-type
    controls [16]. Fibromodulin (FMOD) is an extracellular matrix (ECM) gene
    expressed abundantly in muscle and connective tissues similar to cartilage, pores and skin, and tendons [17] and
    an MSTN regulator that controls muscle cell formation during the myogenic
    program [18].
    The VS./HTS approach is used to display screen compound
    libraries shortly and cost-effectively using
    high-end computational approaches. The actions of many recognized pure compounds have yet to be decided, corresponding to these detailed
    in the Chinese Language conventional medicine and Korean medicinal compound databases.

    Several groups have attempted to identify novel therapeutic candidates that focus on MSTN, but unfortunately, a massive quantity
    of recognized compounds were not MSTN specific and have been also discovered to dam activin A
    and TGF-β signaling (Suh and Lee, 2020a). The
    particular concentrating on of MSTN stays a major analysis challenge as many
    TGF-β ligands exhibit considerable structural similarities.

    Myostatin and activins (A and B) bind to the ActRIIA or ActRIIB receptor on the cell membrane and can signal
    through it. Inhibition of muscle hypertrophy and differentiation by myostatin occurs via Smad2/3 phosphorylation-dependent
    blockade of the AKT-mTOR pathway [180]. So that
    the treatment of muscle with myostatin or activin results in a lower
    within the degree of phosphorylated AKT. Easy blocking of myostatin was one of many first and most evident therapeutic strategies to correct
    muscle atrophy. Because clear hypertrophy was seen in myostatin-null animals and in humans who were null for this ligand.

    Nonetheless, the extent of myostatin reported
    in humans is decrease than the extent noticed in rodents.
    Subsequently, it is affordable to ask the query that
    blocking myostatin alone may be effective or that we
    must always seek to inhibit other ligands that play a task within the adverse regulation of muscle mass by sending signals via the same receptor.

    Additional research are needed to totally probe the mechanisms underlying muscle loss utilizing the hindlimb unloading model.

    But hindlimb suspension is a complex mannequin that mimics sure features
    of spaceflight where muscle contraction and motion can still occur7.

    Hindlimb suspension also creates a head-down tilt situation, causing a cephalic fluid shift similar to that which happens in astronauts.
    Stress is increased through activation of the hypothalamic-pituitary-adrenal (HPA) axis leading to elevated glucocorticoids, primarily corticosterone13.
    Suspended mice lose up to 10% of their body weight within the
    first two days of suspension9. Fasting causes muscle atrophy with out increases in the glucocorticoid corticosterone14,15.

    Primary human myogenic cultures have been derived from skeletal
    muscle biopsies of eight wholesome adults.
    After differentiation, myotubes were treated with zero.002,
    1 and 50 µM UA or automobile for 24 h. Basal and insulin-stimulated glucose uptake was measured using tritiated deoxy-D-glucose, while amino acid-stimulated protein synthesis was estimated utilizing the surface
    sensing of translation (SuNSET) approach. Expression of myostatin and glucose transporters
    was quantified through real-time PCR. FST-related gene (FLRG) also referred to as FSTL3 protein, reveals excessive homology
    to a 10-cysteine repeat of FST.
    Despite the lack of security data, myostatin inhibitors have turn out to be popular
    athletic dietary supplements to use for purported quick muscle
    growth [36]. Myostatin supplements are products designed to
    block or cut back myostatin activity, selling muscle development and combating
    muscle atrophy. Sufficient sleep and recovery intervals are essential in enhancing muscle power and growth by permitting the body to repair and build skeletal muscle mass.
    Spinach is filled with compounds that can stimulate insulin-like development factors, probably reducing serum
    myostatin levels. In animal models, myostatin blockers
    have proven dramatic results, similar to increased muscle measurement and better insulin resistance.
    Scientific studies in humans are nonetheless limited, but
    early results are promising for each athletic and medical functions.
    First, blood samples were collected solely on day 1 (onset of the run-in phase) and day 42 (end of the recovery).

    This ingredient helps in growing the levels of
    follistatin and maintaining myostatin levels low.

    If you need to know tips on how to decrease myostatin blocker, simply remember that there’s an important compound in the muscle-building course of called follistatin. It
    regulates myostatin-signaling pathways, leading to lower motion of myostatin inhibition that
    ends in extra muscle fiber building.
    MSTN negatively impacts bone formation by inhibiting osteogenic differentiation of mesenchymal stem cells and osteoblasts
    [146, 147]. It also suppresses chondrogenesis, delaying the
    transition from cartilage to bone throughout fracture healing,
    thereby affecting callus formation and bone regeneration [148].
    MSTN is proven to be a optimistic regulator of
    osteoclast differentiation, which is answerable for
    the resorption of aged bone and plays a job in bone degradation in arthritis and osteoporosis [149].

    Follistatin, in excess, raises the chance of a number
    of malignancies, including cutaneous, esophageal, stomach, and
    prostate cancer. Nevertheless, excessive ranges of Follistatin have been found to have a preventive impact in opposition to breast cancer, whereas additional
    examine is required.It is essential to know that, like different anabolic steroids, YK11 may cause varied adverse
    effects ranging from minor to extreme. Elevated hair development,
    acne, modifications in libido, sleeplessness, anxiousness, irritability, nausea,
    and headaches are all widespread unwanted facet effects.
    Excessive blood pressure, liver harm, renal harm, and coronary heart harm are all potential opposed effects.Given the hazards, it is
    advised that only those with previous experience with anabolic steroid use consider utilising YK11.
    These contemplating YK11 dietary supplements should be aware of the hazards and proceed with warning.

    Blood sampling is a relatively quick procedure and fewer invasive compared to muscle biopsies that had
    been obtained also at these two additional time points.

    The CSA of vastus lateralis measured by B-module ultrasonography was validated as an acceptable alternative to MRI,
    which is considered the gold standard for measuring muscle measurement, for measuring changes in muscle dimension in response to disuse atrophy [23].
    Utilizing this method, with a 18L5 probe (BK Medical
    North America, Peabody, MA, USA), we measured the CSA of vastus lateralis of the leg randomly assigned for immobilization at days 1, 14, 28, and 42.
    The procedure involved individuals laying supine for more
    than 10 min to normalize fluid shifts in the physique, and we marked the location for imaging, 50% of the gap
    between the top of the patellar bone and the higher trochanter
    of the femur, recognized by palpation. Afterward,
    a straight line was drawn alongside the longitudinal of the vastus lateralis, and the pictures had been taken at 2 cm increments alongside the
    road leading to ~7–9 pictures.
    Regardless, comprehensive analysis using refined methodologies to
    accurately measure serum MSTN concentrations is essential to greatest determine if lowered MSTN impacts
    the effectiveness of inhibition therapies [103, 104].

    Electrical stimulation of the soleus muscle was performed to measure energy, fatigue, and restoration from fatigue following all treatments.
    A detailed description of soleus electrophysiology experiments
    is described in Hanson et al.9.
    This not only aids in muscle restoration and progress but additionally enhances the effectiveness of different dietary supplements by improving their bioavailability.
    This regulatory mechanism ensures that muscle
    progress doesn’t go unchecked, which is crucial for normal development.

    MightyBecause they wished to know for sure whether the Mighty gene is essential to muscle progress the
    researchers did another experiment. The figures beneath show that within the muscle cells where this
    occurred [shRNA Akirin-1] fewer muscle fibres had been indeed fashioned than in muscle cells in which the
    Mighty gene was still energetic [shRNA scrambled].
    Exercise-induced mitochondrial biogenesis happens when skeletal muscle cells create new mitochondria
    in response to strenuous train. As a result of muscle mitochondrial content increasing, basal metabolic fee rises, oxidative capacity expands, and a number of other advantageous variations happen [21].

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